Comcrete Post Spikes

Acid Extractable Matrix Spikes - 1ML

CLPS-MSA15-TI 1ML
EUR 59.4

Human IgG antibody Laboratories manufactures the comcrete post spikes reagents distributed by Genprice. The Comcrete Post Spikes reagent is RUO (Research Use Only) to test human serum or cell culture lab samples. To purchase these products, for the MSDS, Data Sheet, protocol, storage conditions/temperature or for the concentration, please contact Spike S. Other Comcrete products are available in stock. Specificity: Comcrete Category: Post Group: Spikes

Human True insulin ELISA kit

1 plate of 48 wells
EUR 624
Description: A competitive ELISA for quantitative measurement of Human True insulin in samples from blood, plasma, serum, cell culture supernatant and other biological fluids. This is a high quality ELISA kit developped for optimal performance with samples from the particular species.

Human True insulin ELISA kit

1 plate of 96 wells
EUR 822
Description: A competitive ELISA for quantitative measurement of Human True insulin in samples from blood, plasma, serum, cell culture supernatant and other biological fluids. This is a high quality ELISA kit developped for optimal performance with samples from the particular species.

Human True insulin ELISA kit

96T
EUR 700
Description: ELISA

Human True Insulin ELISA Kit

10x96-Strip-Wells
EUR 6725

Human True Insulin ELISA Kit

48-Strip-Wells
EUR 550

Human True Insulin ELISA Kit

5x96-Strip-Wells
EUR 3420

Human True Insulin ELISA Kit

96-Strip-Wells
EUR 765

Spikes information

Handle Post Mortem Knife - EACH

INS4456 EACH
EUR 10.8

Blades Post Mortem PM60B - PK50

INS4459 PK50
EUR 45.9

Blades Post Mortem For INS4456 - PK10

INS4458 PK10
EUR 32.4

Vaginal Swabs, Human Donor - Post-Coitus

MBS170223-INQUIRE INQUIRE Ask for price

Recombinant RSV Post-fusion glycoprotein F0

DAG-WT1180 1 mg
EUR 1360
Description: Recombinant

POST & RUBBER BASE RED PK 4 AC Signs

256730 Pack of 4 Piece(s)
EUR 102.43
Description: Description Dutch: Paal & rubber voetstuk (4 st.); Description French: Poteau & socle en caoutchouc (1,6 kg)

POST & RUBBER BASE WHITE PK 4 AC Signs

256726 Pack of 4 Piece(s)
EUR 102.43
Description: Description Dutch: Paal & rubber voetstuk (4 st.); Description French: Poteau & socle en caoutchouc (1,6 kg)

POST & FILLABLE BASE RED PK 4 AC Signs

256729 Pack of 4 Piece(s)
EUR 94.38
Description: Description Dutch: Paal & opvulbaar voetstuk (4 st.); Description French: Poteau & socle lestable (3,7 litres à remplir)

POST & FILLABLE BASE WHITE PK 4 AC Signs

256725 Pack of 4 Piece(s)
EUR 94.38
Description: Description Dutch: Paal & opvulbaar voetstuk (4 st.); Description French: Poteau & socle lestable (3,7 litres à remplir)

Cerebrospinal Fluid (CSF), Post-Mortem Human Donor

MBS170267-INQUIRE INQUIRE Ask for price

BRADYLINK POST & TRI PLAS BASE RED AC Signs

292233 Pack of 4 Piece(s)
EUR 102.91
Description: Description Dutch: Paal & plastic voetstuk (geen cement); Description French: Poteau & socle en plastique sans béton

PMS2 Antibody / Post Meiotic Segregation Increased 2

V5202-100UG 100ug
EUR 363.3
Description: PMS2 is involved in DNA mismatch repair. It forms a heterodimer with MLH1 and this complex interacts with other complexes bound to mismatched bases. Defects in PMS2 are the cause of hereditary non-polyposis colorectal cancer type 4 (HNPCC4). Mutations in more than one gene locus can be involved alone or in combination in the production of the HNPCC phenotype (also called Lynch syndrome). Most families with clinically recognized HNPCC have mutations in either MLH1 or MSH2 genes. HNPCC is an autosomal, dominantly inherited disease associated with marked increase in cancer susceptibility. It is characterized by a familial predisposition to early onset colorectal carcinoma (CRC) and extra-colonic cancers of the gastrointestinal, urological and female reproductive tracts. HNPCC is reported to be the most common form of inherited colorectal cancer in the Western world, and accounts for 15% of all colon cancers. Defects in PMS2 are a cause of mismatch repair cancer syndrome (MMRCS); also known as Turcot syndrome or brain tumor-polyposis syndrome 1 (BTPS1). MMRCS is an autosomal dominant disorder characterized by malignant tumors of the brain associated with multiple colorectal adenomas. Skin features include sebaceous cysts, hyperpigmented and cafe au lait spots.

PMS2 Antibody / Post Meiotic Segregation Increased 2

V5202-20UG 20ug
EUR 160.3
Description: PMS2 is involved in DNA mismatch repair. It forms a heterodimer with MLH1 and this complex interacts with other complexes bound to mismatched bases. Defects in PMS2 are the cause of hereditary non-polyposis colorectal cancer type 4 (HNPCC4). Mutations in more than one gene locus can be involved alone or in combination in the production of the HNPCC phenotype (also called Lynch syndrome). Most families with clinically recognized HNPCC have mutations in either MLH1 or MSH2 genes. HNPCC is an autosomal, dominantly inherited disease associated with marked increase in cancer susceptibility. It is characterized by a familial predisposition to early onset colorectal carcinoma (CRC) and extra-colonic cancers of the gastrointestinal, urological and female reproductive tracts. HNPCC is reported to be the most common form of inherited colorectal cancer in the Western world, and accounts for 15% of all colon cancers. Defects in PMS2 are a cause of mismatch repair cancer syndrome (MMRCS); also known as Turcot syndrome or brain tumor-polyposis syndrome 1 (BTPS1). MMRCS is an autosomal dominant disorder characterized by malignant tumors of the brain associated with multiple colorectal adenomas. Skin features include sebaceous cysts, hyperpigmented and cafe au lait spots.

PMS2 Antibody / Post Meiotic Segregation Increased 2

V5202SAF-100UG 100ug
EUR 363.3
Description: PMS2 is involved in DNA mismatch repair. It forms a heterodimer with MLH1 and this complex interacts with other complexes bound to mismatched bases. Defects in PMS2 are the cause of hereditary non-polyposis colorectal cancer type 4 (HNPCC4). Mutations in more than one gene locus can be involved alone or in combination in the production of the HNPCC phenotype (also called Lynch syndrome). Most families with clinically recognized HNPCC have mutations in either MLH1 or MSH2 genes. HNPCC is an autosomal, dominantly inherited disease associated with marked increase in cancer susceptibility. It is characterized by a familial predisposition to early onset colorectal carcinoma (CRC) and extra-colonic cancers of the gastrointestinal, urological and female reproductive tracts. HNPCC is reported to be the most common form of inherited colorectal cancer in the Western world, and accounts for 15% of all colon cancers. Defects in PMS2 are a cause of mismatch repair cancer syndrome (MMRCS); also known as Turcot syndrome or brain tumor-polyposis syndrome 1 (BTPS1). MMRCS is an autosomal dominant disorder characterized by malignant tumors of the brain associated with multiple colorectal adenomas. Skin features include sebaceous cysts, hyperpigmented and cafe au lait spots.

PMS2 Antibody / Post Meiotic Segregation Increased 2

V5203-100UG 100ug
EUR 363.3
Description: PMS2 is involved in DNA mismatch repair. It forms a heterodimer with MLH1 and this complex interacts with other complexes bound to mismatched bases. Defects in PMS2 are the cause of hereditary non-polyposis colorectal cancer type 4 (HNPCC4). Mutations in more than one gene locus can be involved alone or in combination in the production of the HNPCC phenotype (also called Lynch syndrome). Most families with clinically recognized HNPCC have mutations in either MLH1 or MSH2 genes. HNPCC is an autosomal, dominantly inherited disease associated with marked increase in cancer susceptibility. It is characterized by a familial predisposition to early onset colorectal carcinoma (CRC) and extra-colonic cancers of the gastrointestinal, urological and female reproductive tracts. HNPCC is reported to be the most common form of inherited colorectal cancer in the Western world, and accounts for 15% of all colon cancers. Defects in PMS2 are a cause of mismatch repair cancer syndrome (MMRCS); also known as Turcot syndrome or brain tumor-polyposis syndrome 1 (BTPS1). MMRCS is an autosomal dominant disorder characterized by malignant tumors of the brain associated with multiple colorectal adenomas. Skin features include sebaceous cysts, hyperpigmented and cafe au lait spots.

PMS2 Antibody / Post Meiotic Segregation Increased 2

V5203-20UG 20ug
EUR 160.3
Description: PMS2 is involved in DNA mismatch repair. It forms a heterodimer with MLH1 and this complex interacts with other complexes bound to mismatched bases. Defects in PMS2 are the cause of hereditary non-polyposis colorectal cancer type 4 (HNPCC4). Mutations in more than one gene locus can be involved alone or in combination in the production of the HNPCC phenotype (also called Lynch syndrome). Most families with clinically recognized HNPCC have mutations in either MLH1 or MSH2 genes. HNPCC is an autosomal, dominantly inherited disease associated with marked increase in cancer susceptibility. It is characterized by a familial predisposition to early onset colorectal carcinoma (CRC) and extra-colonic cancers of the gastrointestinal, urological and female reproductive tracts. HNPCC is reported to be the most common form of inherited colorectal cancer in the Western world, and accounts for 15% of all colon cancers. Defects in PMS2 are a cause of mismatch repair cancer syndrome (MMRCS); also known as Turcot syndrome or brain tumor-polyposis syndrome 1 (BTPS1). MMRCS is an autosomal dominant disorder characterized by malignant tumors of the brain associated with multiple colorectal adenomas. Skin features include sebaceous cysts, hyperpigmented and cafe au lait spots.

PMS2 Antibody / Post Meiotic Segregation Increased 2

V5203SAF-100UG 100ug
EUR 363.3
Description: PMS2 is involved in DNA mismatch repair. It forms a heterodimer with MLH1 and this complex interacts with other complexes bound to mismatched bases. Defects in PMS2 are the cause of hereditary non-polyposis colorectal cancer type 4 (HNPCC4). Mutations in more than one gene locus can be involved alone or in combination in the production of the HNPCC phenotype (also called Lynch syndrome). Most families with clinically recognized HNPCC have mutations in either MLH1 or MSH2 genes. HNPCC is an autosomal, dominantly inherited disease associated with marked increase in cancer susceptibility. It is characterized by a familial predisposition to early onset colorectal carcinoma (CRC) and extra-colonic cancers of the gastrointestinal, urological and female reproductive tracts. HNPCC is reported to be the most common form of inherited colorectal cancer in the Western world, and accounts for 15% of all colon cancers. Defects in PMS2 are a cause of mismatch repair cancer syndrome (MMRCS); also known as Turcot syndrome or brain tumor-polyposis syndrome 1 (BTPS1). MMRCS is an autosomal dominant disorder characterized by malignant tumors of the brain associated with multiple colorectal adenomas. Skin features include sebaceous cysts, hyperpigmented and cafe au lait spots.